Use Nature to Safely and Effectively Lessen and Eliminate Old Scars
by Tony Isaacs
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Few of us go through life without accumulating a few scars. Sometimes those scars can be unsightly and in other instances old scars and scar tissue can cause physical discomfort and other problems. If a person is persistent however, scars can often be greatly diminished and even eliminated naturally. Before opting for cosmetic surgery, chemical peels or botox, consider the following safer options:
The first item to consider is digestive enzymes, which are particularly effective in removing and reducing scar tissue. Scars are formed from fibrin as part of the body's natural repair mechanism. Digestive enzymes dissolve and remove fibrin. Serrapeptase and nattokinase are among the best enzymes for scar tissue removal. Taking the enzymes in quantity 2-3 times daily on an empty stomach gradually dissolves scar tissue. In time even old scar tissue, including keloids, can be dissolved and "eaten away".
For more information, see:
http://www.tbyil.com/Enzymes.htm
Other natural remedies for lessening and eliminating scars are:
*Applying and massaging lavender oil directly on scars helps them fade and in time can completely eliminate scars. Lavender oil also helps newer injuries heal faster with the application of lavender oil.
*Flaxseed oil is a great way to fade scars. You can buy it at almost any health food store. It is also great for new scars, helping them heal faster and keeping them from becoming as noticeable. Rub the flaxseed oil on old and new scars a few times each day, including after you shower and again before going to bed.
*Virgin, unrefined hempseed oil used twice daily often results in improvement and fading within a few weeks
*Grapeseed extract, jojoba and almond oil are all effective scar treatments, especially when combined together.
*Apply vitamin K cream topically to scars. Depending on the severity of your scars and skin type, you may see results in as little as a couple of weeks to a couple of months when used twice daily.
*Apply olive oil (100% extra virgin) to scars at least twice a day. Within a week you should start to see scars and stretch marks starting to fade.
*Organic raw honey (especially high UMF factor manuka honey) rubbed onto scars twice daily will break up scar tissue and help with healing.
*Take finely ground, powder-like, coffee and mix it with one of the oils mentioned here. Rub into scars for about 60 seconds and wipe off the coffee grounds with a warm moist towel. Do this about twice a day if possible. You should see noticeable results in a few weeks. This remedy is especially good for stretch mark scars.
*Use castor oil, plastic wrap and a heating pad. Note: Be sure to use only food grade plastic wrap that contains no bisphenol A, a dangerous substance found in many plastics. Rub plenty of castor oil on the scars and then wrap it with plastic wrap. Place a heating pad turned on as high a setting as you comfortably stand and leave on for 20 to 25 minutes. You may see noticeable results in as little as a week or so.
*Use vitamin E oil from punctured gel caps and rub it into scars.
* Colloidal Silver. Colloidal silver is especially good on healing wounds. It aids in tissue repair and can result in significantly lessened scarring.
*Massaging your scars often and consistently will help break up the fibrous tissue and can be especially effective when combined with one of the oils mentioned above.
Note: The key to natural lessening and elimination of scar tissue is persistence. Serious scars may take up to several months, but with persistent and consistent effort you should eventually see good results.
Sources included:
http://www.naturalnews.com/028285_enzymes_scars.html
http://www.tbyil.com/Scars_and_Stretch_Marks.htm
http://www.natural-homeremedies.com/...ssential-oils/
About the author:
Tony Isaacs is a natural health advocate and researcher and the author of books and articles about natural health including Cancer's Natural Enemy. Mr. Isaacs is a contributing author for SANEVAX and his articles are regularly featured at Natural News, the Health Science Institute's Healthiertalk website, CureZone, the Crusador online, AlignLife, the Cancer Tutor, the American Chronicle and several other venues. Mr. Isaacs also has The Best Years in Life website for baby boomers and others wishing to avoid prescription drugs and mainstream managed illness and live longer, healthier and happier lives naturally. In addition, he hosts the Yahoo Oleandersoup Health group of over 2000 members and the CureZone "Ask Tony Isaacs - Featuring Luella May" forum.
Use Enzymes for Fibrosis, Scars, Keloids, Lung Disease and Cancer
by Tony Isaacs
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A primary benefit of natural enzymes is eliminating and preventing scar tissue and fibrosis. Fibrosis itself is scar tissue. As we age, fibrosis becomes a leading cause of eventual death. Around our late 20's our bodies being to produce a diminishing output of enzymes and by the time we reach our 40's we produce very little enzymes. Cystic Fibrosis patients have virtually no enzyme production to speak of, and even cystic fibrosis children seldom make it past their 20's before they die of the restriction and shrinkage in the lungs from the formation of fibrosis or scar tissue.
As a result of diminishing enzyme production, our bodies are less and less able to reduce the over abundance of fibrin it deposits in nearly everything from simple cuts, to the inside of our internal organs and blood vessels. This inability is particularly noticeable in women, who frequently develop conditions such as fibrocystic breast disease, uterine fibroids, adhesions, and endometriosis. Men and women alike grow arterial sclerotic plaque (which is scar tissue). We are all also prone to have fibrin begin to spider its way into our internal organs, reducing the organs size and function over time. As we age, our wounds heal with thicker, less pliable, weaker and more visible scars.
By replacing lost enzymes we can help control and reduce scar tissue and fibrosis, thus preventing many of the problems associated with lack of enzymes and excess fibrin. Adding vital enzymes can also help reverse and control existing conditions caused by fibrosis. Even old scar tissue can be dissolved and "eaten away" from surgical wounds, pulmonary fibrosis, kidney fibrosis and even keloids years after their formation. Though doctors in the United States are only recently becoming aware of the benefits of added enzymes, doctors in Europe and Asia have been administering enzymes for decades.
Among the best enzymes for removal of scar tissue are serrapeptase and nattokinase. Chymotypsin and pancreatin are considered to be especially valuable against cancer tumors, especially pancreatic cancer tumors.
An especially powerful enzyme is serrapeptase, which is a proteolytic enzyme derived from a species of bacteria originally found in the intestines of silkworms. The bacteria Serratia mercesans E1 produces Serrapeptase, an enzyme that enables the silkworm to dissolve its silken cocoon and emerge after metamorphosis. Serratia is produced by fermented cultures. Studies have shown that serrapeptase reduces scar tissue, improves tissue healing and significantly reduces inflammation.
Serrapeptase also has the unique ability to digest non-living tissue that is a by-product of the healing response without harming living tissue.
Serrapeptase is used to dissolve non-living tissues to include: scar tissue, fibrosis, blood clots, cysts and arterial plaque. It is also used as an anti-inflammatory agent against Sinusitis and for thinning mucous secretion and as an agent against varicose veins and fibrocystic breasts. It is believed that serrapeptase can play a key role in dissolving the outer protective layers of cancer cells and tumors to enable the immune system and other cancer fighters to better attack the cancer.
Serrapeptase may be particularly effective for those who have lung problems, as it clears out all of the inflammation, mucus and dead/scar tissue. Clearing away problem tissues enables the body's own natural healing system to be able to better replace the bad tissue with healthy tissue and result in better lung function. Serrapeptase also helps eliminate old scar tissue by slowly digesting all of the dead tissue/scar tissue so that it can be replaced as needed with healthy tissue
Serrapeptase is particularly sensitive to a low pH, thus the supplement versions which are enterically coated are considered superior.
Sources included:
http://www.articlesnatch.com/Article...e-Blood/458516
http://www.thewomenwarriors.net/phpb...opic.php?t=414
www.dr-gonzalez.com/clinical_pearls.htm
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What does Serrapeptase do?
Serrapeptase is a powerful proteolytic enzyme formula that stands alone in fighting the combined threats of fibrin, mucous and inflammation in the body.
Serrapeptase is a protein digesting enzyme which breaks down "non living" matter in the human body. This may promote a reduction in levels of dead tissue in the circulatory system, promoting smoother healthier flowing blood. Fibrin often builds up throughout the body and manifest in many undesirable and unhealthy ailments. Serrapeptase fights fibrin build up in the cardiovascular system, organs and muscle tissue.
The late German physician, Dr. Hans Nieper, used Serrapeptase to treat arterial blockage in his coronary patients. He found that Serrapeptase helps against stroke and is reportedly more effective and quicker than EDTA Chelation treatments in removing arterial plaque. He also reports that Serrapeptase dissolves blood clots and causes varicose veins to shrink or diminish. Dr. Nieper told of a woman scheduled for hand amputation and a man scheduled for bypass surgery who both recovered quickly without surgery after treatment with Serrapeptase .
Studies and reports on the use of Serrapeptase are numerous and reports reference successful use with:
Dissolving non-living tissues to include blood clots, cysts and arterial plaque
As an anti-inflammatory agent
Sinusitis and for thinning mucous secretions
Removing Arterial Blockages
Rheumatoid Arthritis
Arthritis Pain and Inflammation
Varicose Veins
Fibrocystic Breasts
Serrapepetase - is a powerful proteolytic enzyme formula that stands alone in fighting the combined threats of fibrin, mucus and inflammation in the body. SerraRX80 is infused serrapeptase alone, no excipients or other products to obstruct, inhibit or in any other way negatively affect results.*
Serrapeptase - noun, ser·ra·pep·tase; a super protein digesting enzyme which breaks down "non living" matter in the human body. This action may promote a reduction in levels of dead tissue in the circulatory system, promoting smoother, healthier flowing blood and reduction of inflammation. Fibrin may build up throughout the body and manifest in many undesirable and unhealthy ailments including abnormal mucus build-up. Serrapeptase fights fibrin build-up in the cardiovascular system, organs, muscle tissue, and throughout the entire human body.*
A Potent Proteolytic Enzyme:
The inflammatory response is an important mechanism for protecting the body from attack by invading organisms and faulty cells. In the case of immune dis-regulation, the body loses its ability to differentiate between innocuous and potentially dangerous substances. This defective mechanisms results in a wide array of autoimmune diseases such as allergies, psoriasis, rheumatoid arthritis, ulcerative colitis, uveitis, multiple sclerosis and some forms of cancer.
Standard drug therapy for inflammatory-mediated diseases and trauma include steroids and non-steroidal anti-inflammatory agents (NSAIDs). Both classes of drugs offer temporary, symptomatic relief from swelling, inflammation and accompanying pain without treating the underlying condition. These drugs may also be immunosuppressive and cause dangerous side effects. The conscientious physician must weigh the benefits and long-term risks associated with the use of NSAIDs, especially in cases of rheumatoid arthritis. If left untreated, the inflammatory process itself can lead to limitation of joint function and destruction of bone, cartilage and articular structures.
NSAIDs are among the most widely prescribed drugs for rheumatoid arthritis and other inflammatory joint conditions. Their effects are mediated through inhibition of the biosynthesis of prostaglandins. They work by irreversibly blocking cyclooxygenase, the enzyme which catalyses the reactions of arachidonic acid to endoperoxide compounds.
The neurological and gastrointestinal side effects of these agents have been reviewed in considerable detail. All of the NSAIDs, with the exception of Cytotec, inhibit prostaglandin El, a local hormone responsible for gastric mucosai cytoprotection. A common side effect from these medications is gastric ulcers. More serious adverse reactions such as blood dyscrasias, kidney damage and cardiovascular effects have been noted. Most physicians rotate among the ten most widely prescribed NSAIDs, as soon as one causes side effects or stops working.
The search for a physiologic agent that offers anti-inflammatory properties without causing side effects may have ended with the discovery of the Serratia peptidase (SP) enzyme. This anti-inflammatory agent is in wide clinical use throughout Europe and Asia as a viable alternative to salicylates, ibuprofen (sold as an OTC in the U.S.) and the more potent NSAIDs. Unlike these drugs, SP is a naturally occurring, physiologic agent with no inhibitory effects on prostaglandins and devoid of gastrointestinal side effects.
SP is an anti-inflammatory, proteolytic enzyme isolated from the microorganism, Serratia El5. This enzyme is naturally present in the silkworm intestine and is processed commercially today through fermentation. This immunologically active enzyme is completely bound to the alpha 2 macroglobulin in biological fluids. Histologic studies reveal powerful anti-inflammatory effects of this naturally occurring enzyme.
The silkworm has a symbiotic relationship with the Serratia microorganisms in its intestines. The enzymes secreted by the bacteria in silkworm intestines have a specific affinity to avital tissue and have no detrimental effect on the host's living cells. By dissolving a small hole in the ~ silkworm's protective cocoon (avital tissue), the winged creature is able to emerge and fly away.
The discovery of this unique biological phenomenon led researchers to study clinical applications of the SP enzyme in man. In addition to its widespread use in arthritis, fibrocystic breast disease and carpal tunnel syndrome, researchers in Germany have used SP for atherosclerosis. SP helps to digest atherosclerotic plaque without harming the healthy cells lining Z the arterial wall. Today, researchers consider atherosclerosis an inflammatory condition similar to other degenerative diseases. Some immunologists are even categorizing atherosclerosis as a benign tumor.
Hardening and narrowing of the arterial wall is a cumulative result of microscopic trauma; inflammation occurs in the presence of oxidized lipids. SP doesn't interfere with the synthesis of cholesterol in the body, but helps clear avital tissue from the arterial wall. It is important to note that cholesterol in its pure state is an antioxidant and a necessary component of the major organ systems in the body. The use of medications, which block cholesterol biosynthesis, may eventually damage the liver and compromise anti-oxidant status of the eyes, lungs and other soft tissues.
While studies with Serrapeptase in the treatment of coronary artery disease are relatively new, a wealth of information exists regarding its anti-inflammatory properties. SP has been used as an anti-inflammatory agent in the treatment of chronic sinusitis, to improve the elimination of bronchopulmonary secretions, traumatic injury (e.g. sprains and torn ligaments), post-operative inflammation and to facilitate the therapeutic effect of antibiotics in the treatment of infections. In the urological field, SP has been used successfully for cystitis and epididymitis.
SP has been admitted as a standard treatment in Germany and other European countries for the treatment of inflammatory and traumatic swellings. In one double-blind study of SP conducted by Esch et al at the German State Hospital in UIm, 66 patients with fresh rupture of the lateral ligament treated surgically were divided in three randomized groups. In the group receiving the test substance, the swelling had decreased by 50% on the third post-operative day, while in the other two control groups (elevation of the leg, bed rest, with or without the application of ice), no reduction in swelling had occurred at that time.
The difference was of major statistical significance. Decreasing pain correlated for the most part with the reduction in swelling. The patients receiving SP became pain-free more rapidly than the control groups. By the 10th day, all patients were free of pain in the SP-treated group. The therapeutic daily dose was 1-2 tablets (5 mg) 3 times daily. In another double-blind study, the anti-inflammatory enzyme, SP, was evaluated in a group of 70 patients with evidence of cystic breast disease. These patients were randomly divided into a treatment group and a placebo group. SP was noted to be superior to placebo for improvement of breast pain, breast swelling and induration with 85.7% of the patients receiving SP reporting moderate to marked improvement. No adverse reactions were reported with the use of SP. The use of enzymes with fibrinolytic, proteolytic and anti-edemic activities for the treatment of inflammatory conditions of the ear, nose and throat has gained increasing support in recent years.
In a third double-blind study, 193 subjects suffering from acute or chronic ear, nose or throat disorders were evaluated. Treatment with SP lasted 7-8 days, two 5 mg tabs, t.i.d. After 3-4 days treatment, significant symptom regression was observed in the SP-treated group, while this was not noted in the control group. Patients suffering from laryngitis, catarrhal rhinopharyngitis and sinusitis noted markedly rapid improvement.
The physicians' assessments of efficacy of treatment were excellent or good for 97.3% of patients treated with SP compared with only 21.9% of those treated with placebo. In a similar study of chronic bronchitis, conducted by a team of otolaryngolosits, the SP-treated group showed excellent results compared with the placebo group in the improvement of loosening sputum, frequency of cough and expectoration. Other improvements included the posterior nasal hydro rhea and rhinos enosis. The administration of SP reduces the viscosity of the nasal mucus to a level at which maximal transport can be achieved. It has also been demonstrated that the simultaneous use of the peptidase and an antibiotic results in increased concentrations of the antibiotic at the site of the infection.
The mechanisms of action of SP, at the sites of various inflammatory processes consist fundamentally of a reduction of the exudative phenomena and an inhibition of the release of the inflammatory mediators. This peptidase induces fragmentation of fibrinose aggregates and reduces the viscosity of exudates, thus facilitating drainage of these products of the inflammatory response and thereby promoting the tissue repair process.
Studies suggest that SP has a modulatory effect on specific acute phase proteins that are involved in the inflammatory process. This is substantiated by a report of significant reductions in C3 and C4 complement, increases in opsonizing protein and reductions in concentrations of haptoglobulin, which is a scavenger protein that inhibits lysosomal protease. Carpal tunnel syndrome is a form of musculol-igamentous strain caused by repetitive motion injury. Individuals who work at keyboard terminals are particularly susceptible to this condition.
While surgery has been considered the first line treatment for carpal tunnel syndrome, recent studies reveal that the use of anti-inflammatory enzymes (e.g. SP and bromelain) in conjunction with vitamins B2 and B6 are also effective. The use of non-invasive, nutritional approaches to the treatment of this common condition will become more important as a generation of keyboard operators approach retirement. Several research groups have reported the intestinal absorption of SP. SP is well absorbed orally when formulated with an enteric coating. It is known that proteases and peptidases are only absorbed in the intestinal area.
These enzymes are mobilized directly to the blood and are not easily detectible in urine. Other enzymes with structural similarities have been reported to be absorbed through the intestinal tract. Chymotrypsin is transported into the blood from the intestinal lumen. Horseradish peroxidase can cross the mucosal barrier of the intestine in a biologically and immunologically active form. Several studies have appeared so far which refer to the systemic effects of orally given proteases and peptidases (e.g. SP), such as repression of oedema and repression of blood vessel permeability induced by histamine or bradykinin. These enzymes also affect the kallikrein-kinin system and the complement system, thus modifying the inflammatory response. In vitro and in vivo studies reveal that SP has a specific, anti-inflammatory effect, superior to that of other proteolytic enzymes. A review of the scientific literature, including a series of controlled, clinical trials with large patient groups, suggests that Serrapeptase is useful for a broad range of inflammatory conditions.
If one considers the fact that anti-inflammatory agents are among the most widely prescribed drugs, the use of a safe, proteolytic enzyme such as SP would be a welcome addition to the physician's armamentarium of physiologic agents.
References:
Serrapeptase references:
l. Kee WH. Tan SL, Lee V. Salmon YM. The treatment of breast engorgement with Serrapeptase (Danzen): a random ized double-blind controlled trial. Singapore Med J. 1989:30(1):48-54.
2. M izukoshi, D. et al. A double-blind clinical study of serrapeptase in the treatment of chronic sinusitis. Igaku Ayrni 109:50-62.1979.
3. Carratu, L. et al. Physio-chemical and rheological research on mucolytic activity of serrapeptase in chronic broncho-pneumopathies. Curr. Ther. Res. 28(6):937-951. 1980.
4. Braga, P.C. et al. Effects of serrapeptase on muco-ciliary clearance in patients with chronic bronchitis. Curr. Ther. Res. 29(5):738-744,1981.
5. Mazzonie, A. et al. Evaluation of serrapeptase in acute or chronic inflammation of otorhinolaryngology pathology: a multicentre, double-blind randomized trial versus placebo. J. int. Med. Res. 18(5):379-388,1990.
6. Kakinumu, A. et al. Regression of fibrinolysis in scalded rats by administration of serrapeptase. Biochem. Pharmacol. 31:2861-2866,1982.
7. Marly, M. Enzymotherapie anti-inflammatoire a l'aide de la serrapeptase: resultats cliniques en traumatologie et en ORL. C RTherapeut. 3:9-19,1985.
8. Odagiri, J. et al. Clinical applications of serrapeptase in sinusitis. Med. Consult. New Remedy 6:201-209, 1979.
9. Yamazaki, J. et al. Anti-inflammatory activity of TSP, a protease produced by a strain of Serratia. Folia Pharmacol. Japon. 6^302-314,1967.
I0. Harad~, Y. Clinical efficacy of serrapeptase on buccal swelling after radical operation for chronic sinusitis. Igaku Ayumi 123:768-778.1982.
11. Matsudo, A. et at. Effect of serrapeptase (Danzen) on inflammatory edema following operation for thyropid disease. Med. Consult. New Remedy 18:171-175, 1981.
12. Fujitani, T. et al. Effect of anti-inflammatory agent on transfer of antibiotics to the maxillary sinus mucosa in chronic sinusitis. Otorhinolaryngol. Clin. North Am. 66:557-565. 1976.
13. Tago. T. and Mitsui, S. Effects of serrapeptase in dissolution of sputum, especially in patients with bronchial asthma. Jap. Clin. Exp. Med. 49:222-228, 1972.
14. Mazzonie, A. et al. Evaluation of serrapeptase in acute or chronic inflammation of otorhinolaryngology pathology: a multicentre, double-blind randomized trial versus placebo. J. int. Med. Res. 18(5):379-388,1990.
15. Kase, Y. et al. A new method for evaluating mucolytic expectorant activity and its application. II. Application to two proteolytic enzymes, serrapeptase and seaprose. Arzneimittelforschung 32:374-378,1982.
16. Marriott, C. Modification of the rheoloaical properties of mucus by drugs. Adv. Exp. Med. Biol. 144^75-84, 1982.
17. Majima. Y. et al. Effects of orally administered drugs on dynamic viscoelasticity of human nasal mucus. Am. Rev. Respit. Dis. 141:79-83.1990.
18. Miyata, K. Intestinal absorption of serrapeptase. J ApplBiochem. 1980:2:111-16.
19. Aso T. et al. Breast engorgement and its treatment: Clinical effects of Danzen (serrapeptase) an anti-inflammatory enzyme preparation. The world of Obstetrics and Gynecology (Japanese). 1981:33:371-9.
20. Esch PM, Gemgross H. Fabian A. Reduction of postoperative swelling. Objective measurement of swelling of the upper ankle joint in treatment with serrapeptase-a prospective study (German). FortschrMed. 1989;107(4):67-8, 71-2.
21. Majima Y, lnagaki M, Hirata K. Takeuchi K, M orishita A, Sakakura Y. The effect of an orally administered proteolytic enzyme on the elasticity and viscosity of nasal mucus. Arch Otorhinolaryngol. 1988;244(6):355-9.
22. Selan L, Berlutti F, Passariello C. Comodi-Ballanti MR, Thaller MC. Proteolytic enzymes: a new treatment strategy for prosthetic infections? Antimicrob Agents Cheroother. 1993; 37(12):2618-21.
23. Koyama A, Mori J, Tokuda H, Waku M, Anno H, Katayama T, Murakami K, Komatsu H, Hirata M, Arai T, et al. Augmentation by serrapeptase of tissue permeation by cefotiam (Japanese). Jpn JAntibiot. 1986; 39(3):761-
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